A maximum likelihood QTL analysis reveals common genome regions controlling resistance to Salmonella colonization and carrier-state

Table 1 Quality data about the SNP markers flanking QTL for Salmonella colonization[7]added to the original SNP data set[4]

Marker	Chr¹	Position (bp)	N²	Inf³	SD⁴	p_SD⁵
IAH-2B	2	19,869,778	182	23/23	NS	0.052
IAH-2 C		22,726,015	182	23/23	S*	0.010
IAH-2D		24,308,758	179	22/23	NS	0.130
IAH-3A	3	92,315,616	152	20/23	S***	0.000
IAH-3B		95,968,986	182	23/23	S***	0.001
IAH-3 C		110,245,601	184	23/23	NS	0.378
IAH-12B	12	14,781,290	181	23/23	NS	0.597
IAH-12 C		17,496,361	181	23/23	NS	0.704
IAH-12D		18,927,306	162	1/23	S***	0.000
IAH-25A	25	875,498	92	17/23	NS	0.093
IAH-25B		1,843,484	184	23/23	NS	0.556

¹Chromosome.
²Number of genotypes available in the F2 Nx6₁ progeny P2.
³Informativity: number of heterozygous F1 parents on the total number of F1 parents.
⁴Significance level of the segregation distorsion evaluated with a Chi-square test: NS non-significant; * significant with p < 0.05 ; *** significant with p < 0.001.
⁵Probabilities associated with a Chi-square test between genotypic frequencies expected according to Mendelian rules and observed frequencies.

ISSN: 1471-2164