Distinctive expansion of gene families associated with plant cell wall degradation, secondary metabolism, and nutrient uptake in the genomes of grapevine trunk pathogens

Table 2 Assembly statistics of the grapevine trunk pathogen genomes analyzed

Species¹	Disease	Assembly size (Mb)	Scaffolds	Scaffold N50 length (Kb)	Scaffold L50 count	Gene space completeness² (%)	Reference
Dia. ampelina (DA912)	Phomopsis dieback³	47.4	2,392	132.3	103	94.8	This work
Dip. seriata (DS831)	Botryosphaeria dieback	37.1	695	304.2	39	96.0	This work
P. chlamydospora (UCR-PC4)	Esca	27.5	702	178.6	50	95.6	This work
E. lata (UCR-EL1)	Eutypa dieback	54.0	2,334	68.3	239	93.2	[23]
T. minima (UCR-PA7)	Esca	47.5	624	334.6	45	95.2	[24]
N.parvum (UCR-NP2)	Botryosphaeria dieback	42.6	1,297	83.6	149	97.2	[25]
F. mediterranea (MF3/22)	Esca complex	63.4	1,412	4,291.5	6	96.4	[37]
S. hirsutum (FP-91666)	Esca complex	46.5	159	1,799.0	9	96.4	[37]

¹The name of the isolate that was sequenced is shown in parenthesis
²Based on CEGMA analysis [40]. Percentages refer to complete KOGs mapped onto the scaffolds
³ Dia. ampelina is also the causal agent of Phomopsis cane and leaf spot, a fruit and foliar disease that affects green tissues

ISSN: 1471-2164